博士,青年研究员,博士生导师
地址:东安路131号
电话:
邮箱:fengweijun@fudan.edu.cn
工作经历
1、Feng, W., Herbst, L., Lichter, P., Pfister, S. M., Liu, H. K., & Kawauchi, D. (2018). CRISPR-mediated loss of function analysis in cerebellar granule cells using in utero electroporation-based gene transfer. Journal of visualized experiments: JoVE, (136).
2、Feng, W*., Shao, C., Liu HK. (2017) Versatile Roles of the Chromatin Remodeler CHD7 during Brain Development and Disease. Frontier Mol. Neuroscience 10:309
3、 Feng, W., Liu HK., Kawauchi D. (2017) CRISPR-engineered genome editing for the next generation neurological disease modeling. Progress in Neuropsychopharmacology & Biological Psychiatry 81:459
4、 Feng, W#., Kawauchi, D#., Korkel-Qu, H., Deng, H., Serger, E., Sieber, L., Lieberman, J.A., Jimeno-Gonzalez, S., Lambo, S., Hanna, B.S., et al. (2017). Chd7 is indispensable for mammalian brain development through activation of a neuronal differentiation programme. Nat Commun 8, 14758.
5、 Feng, W., and Liu, H.K. (2017). Revealing the Hidden Powers that Fuel Adult Neurogenesis. Cell Stem Cell 20, 154-156.
6、 Feng, W., Liu HK. (2013). Epigenetic regulation of neuronal fate determination: The role of CHD7. Cell Cycle 12 (24): 3707-3708
7、Feng, W., Khan, M.A., Bellvis, P., Zhu, Z., Bernhardt, O., Herold-Mende, C., and Liu, HK. (2013). The Chromatin Remodeler CHD7 Regulates Adult Neurogenesis via Activation of SoxC Transcription Factors. Cell Stem Cell 13, 62-72.
8、Xie, W., Ling, T., Zhou, Y., Feng, W., Zhu, Q., Stunnenberg, H.G., Grummt, I., and Tao, W. (2012). The chromatin remodeling complex NuRD establishes the poised state of rRNA genes characterized by bivalent histone modifications and altered nucleosome positions. Proc Natl Acad Sci U S A, 109, 8161-8166.
9、Feng, W#., Yonezawa, M#., Ye, J., Jenuwein, T., and Grummt, I. (2010). PHF8 activates transcription of rRNA genes through H3K4me3 binding and H3K9me1/2 demethylation. Nat Struct Mol Biol 17, 445-450.
10、Yuan, X., Feng, W., Imhof, A., Grummt, I., and Zhou, Y. (2007). Activation of RNA polymerase I transcription by cockayne syndrome group B protein and histone methyltransferase G9a. Molecular Cell 27, 585-595.